CD73 Plays a Protective Role in Collagen-Induced Arthritis

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CD73 plays a protective role in collagen-induced arthritis.

Rheumatoid arthritis (RA) is a chronic autoimmune disease with significant morbidity and mortality. Recent studies suggest that modulation of adenosine signaling, a potent immunosuppressive pathway, is a promising approach for treatment of RA. Extracellular adenosine can come from two sources: transport of intracellular adenosine and hydrolysis of extracellular adenine nucleotides by CD73. In t...

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NLRP3 Inflammasome Plays an Important Role in the Pathogenesis of Collagen-Induced Arthritis.

OBJECTIVE To investigate the relationship between NLRP3 and the pathogenesis of collagen-induced arthritis. METHODS We used the collagen-induced arthritis (CIA) mouse model. The mice were divided into two groups: the model group (CIA, n = 16) and the control group (Normal, n = 8). The mice were sacrificed seven weeks after immunization. The arthritis score and imaging evaluation (X-rays, Micr...

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T Cell Vaccination as a Tool in the Treatment of Collagen Induced Arthritis in Albino Rats

Background: The effectiveness of T cell vaccination has been demonstrated in a variety of animal models of both induced and spontaneous autoimmune diseases. Objective: The purpose of this study was to test the T cell vaccination protocol to treat and prevent collagen induced arthritis (CIA) in a rheumatoid arthritis model. Methods: CIA was induced by an intradermal injection of an artheritogen ...

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Protective role of major histocompatibility complex class II Ebd transgene on collagen-induced arthritis

Collagen-induced arthritis (CIA) is an animal model of autoimmune inflammatory polyarthritis that has features similar to rheumatoid arthritis (RA). Much like RA, susceptibility to mouse CIA is influenced by the major histocompatibility complex (MHC), H-2, and restricted to the H-2q and H-2r haplotypes. Whereas the role of the H-2A molecule in susceptibility to CIA is well established, little i...

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ژورنال

عنوان ژورنال: The Journal of Immunology

سال: 2015

ISSN: 0022-1767,1550-6606

DOI: 10.4049/jimmunol.1401416